Thursday, 4 June 2026

9 Tips for Camping and Hiking With Type 1 Diabetes

From everydayhealth.com

By Jessica Freeborn

If you have type 1 diabetes (T1D), hiking and camping can be a little more complicated. When you’re off in the wilderness, miles from doctors and roads, you’ll need to take special care of your insulin and medical equipment, and complications like low blood sugar (hypoglycaemia) can be especially dangerous. 

“Hiking can bring in so many extra factors to the mix that you might not be expecting when you're doing other types of exercising,” says the certified diabetes care and education specialist Jen Hanson, the executive director of Connected in Motion, a non-profit that organizes wilderness adventures for adults with type 1 diabetes. 

With a little extra planning, says Hanson, who has type 1 diabetes herself, you can have an enjoyable and safe time in the great outdoors.  

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1. Bring Backups for All Your Diabetes Supplies

It’s often recommended to double the amount of diabetes supplies you carry when traveling, and hiking and camping trips are no exception. For people with T1D who have an insulin pump, it may also be a good idea to bring a backup way to deliver insulin.

“Bring both long- and short-acting insulin, syringes or pen needles, and a backup glucagon kit,” says Rekha Kumar, MD, an endocrinologist at NewYork-Presbyterian and Weill Cornell Medicine. She also notes that people who use an insulin pump should bring backup insulin in case of pump malfunctions. 

“Some of the things that I see most commonly when hiking with people with diabetes are [CGM or pump] sites that get ripped out when backpacks are taken off or put on, [and] a lot of sweat that can lead to sites falling off,” Hanson says. 

2. Bring More Sugar Than You Think You’ll Need

Physical activity like hiking increases the risk of low blood sugar, and there are no grocery stores in the wilderness.

“It's probably a good idea to pack more [sugar] than you're going to need,” says Hanson, explaining that hikes can take longer than you expect. She encourages people to think about “the maximum duration of time that you might possibly be out there and pack accordingly.” 

Dr. Kumar adds that it can be a good idea to pack multiple forms of sugar, and you should also consider the environment where you’re hiking. Fruit gummies can freeze in cold weather, and glucose tablets can degrade or clump because of sweat and heat. “Bring gel packets because they’re more reliable in outdoor temperatures,” she says.

3. Inform Friends About Your Condition

Hiking partners play an important role in recognizing and responding to low blood sugar. “The companions that somebody is hiking or camping with should know what low blood sugar or hypoglycaemia looks like,” says Kumar. 

Visible symptoms of hypoglycaemia include:
  • Shakiness
  • Sweating
  • Confusion
  • Weakness
  • Vision changes
  • Slurred speech

Kumar says very low blood sugar may look like intoxication or altitude sickness, and just knowing there's a range of symptoms can be crucial in remote settings where quick recognition matters. 

It’s equally important your camping and hiking partners know how to respond during a low-blood-sugar event. Hanson says friends should understand in advance how you want them to help, including where your low blood sugar supplies are stored. Your friends should be prepared to find your sugar source and help you eat or drink it.

4. Pack Glucagon 

Glucagon is a medication used to treat low blood sugar levels so severe that you can no longer treat them yourself by simply eating or drinking something sugary. It works by signalling the liver to release stored glucose into the bloodstream. 

There are different forms of glucagon available, including a nasal spray or a pen that’s similar to an EpiPen, which Kumar says are preferable in outdoor settings, because they’re faster and easier to administer.

Ideally, you’d have someone on your trip that knows how to properly administer glucagon. “If the person is unable to swallow or loses consciousness, administering glucagon and contacting emergency services can be lifesaving. Clear, simple instructions shared in advance can support a quick and effective response,” says Barbara Eichorst, RD, CDCES, the vice president of healthcare programs at the American Diabetes Association.  

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5. Wear a Medical ID Bracelet

Medical ID bracelets or necklaces alert emergency personnel to conditions such as type 1 diabetes. An ID bracelet could help in a situation where you become unresponsive, such as with extremely low blood sugar.

“Wearing a medical identification bracelet or necklace that indicates type 1 diabetes and insulin use is strongly recommended,” says Eichorst. “In the event of an emergency, this can help others, including first responders, quickly understand the situation and provide appropriate care.” 

6. Adjust Insulin for Activity 

If you’re going to be walking, hiking, kayaking, or skiing for most of the day, you might need to dial down the amount of insulin you’re using, both before and after your workout.

“Aerobic exercise like hiking lowers blood sugar, and that can persist for 12 to 24 hours post exercise. Even the night after you hike, you're at risk of low blood sugar,” says Kumar. For people using insulin pumps, this may involve temporarily reducing basal insulin delivery, while those on injections may need to adjust both long-acting and mealtime doses, she says. 

Hanson says it may be helpful to “think about putting your basal rates back to normal about 30 minutes before you're about to end your hike to avoid a really big blood sugar spike at the end of the day.”

If you’re not familiar with making such adjustments on your own, it’s a good idea to discuss your plans with your doctor. 

7. Stay Hydrated and Fuelled

Consuming enough water is important when exercising and in hot and humid conditions. “Hydration is often overlooked, but is really key, and that's whether you have diabetes or not,” Hanson says. “So don't miss those opportunities to fill your water bottle.”

Don’t neglect proper nutrition either. “During the day, your body is using all the food you're eating for energy, but it's also breaking down energy that's been stored in your muscles for emergencies or just for long days like this,” says Hanson. “And those stores need to be replenished.”

It can be tricky to decide what to eat during a long day of exercise, because you have to contend with more blood sugar variables than normal. “Combining fast-acting carbohydrates for immediate needs with more sustained sources of energy, such as snacks that include carbohydrates and protein, can support stability,” says Eichorst, noting that “eating small amounts at regular intervals during longer hikes can also help maintain more consistent glucose levels.”

8. Take Care of Your Feet

A long hike can lead to foot problems for anyone, and it’s especially important to be proactive about foot health when you have diabetes, since nerve damage (peripheral neuropathy) can occur in both type 1 and type 2 diabetes. This nerve dysfunction can put people at a greater risk for injury and foot infections.

You can prevent blisters by choosing the right shoes and aggressively treating your feet as soon as you feel irritation. “If you're planning on buying a new pair of hiking boots, make sure you give yourself a lot of time to work them in, because a new boot on a long trail on a hot day is a recipe for blisters,” says Hanson. “Stop as soon as you feel rubbing or something feeling off, because fixing it immediately is a great way to stop blisters from happening.” 

When blisters do happen, prompt treatment is critical. “If a blister develops, keeping it clean and covered and monitoring for signs of infection can help support healing,” says Eichorst. 

9. Know When to Call It Quits

In some circumstances, it might be safest to cut a camping or hiking trip short. Kumar says the following scenarios may warrant ending a trip early. 

It may also be important to consider your supplies. With repeated episodes of low blood sugar, “you may have consumed all or most of the treatments that you've brought along with you,” says Hanson. “And you still need emergency treatment for the way out.” 

With higher blood sugars, she says it’s possible to “get to a point where you might have used all of your pump sites, or your insulin has gone bad,” in which case ending a trip is the wisest choice. 

The Takeaway

  • Hiking and camping with type 1 diabetes requires extra preparation: packing backup insulin, glucose sources, and diabetes supplies in case of emergencies or equipment failure.
  • Physical activity, heat, altitude, and changing meal schedules can all affect blood sugar levels, making it important to monitor glucose closely and adjust insulin, hydration, and nutrition as needed.
  • Let hiking companions know how to recognize and respond to low blood sugar, and don’t hesitate to end a trip early if blood sugar becomes unsafe or supplies run low.

Wednesday, 3 June 2026

What Is Type 3 Diabetes?

From goodrx.com 

Key takeaways:

  • Type 3 diabetes is a term that describes the effects of diabetes in the brain. 

  • People with diabetes have a higher risk of dementia. For some, the risk for dementia and diabetes may both be linked to their genetics.

  • Caring for your diabetes is the best way to prevent or slow the progression of Type 3 diabetes, or diabetes-related dementia.

Many people know about Type 1 and Type 2 diabetes. But maybe you’ve also heard the term “Type 3 diabetes.” It’s not a new kind of diabetes, or a diagnosis you will see in your medical chart. It’s a term that describes the effects of diabetes on the brain. 

Here, we’ll talk about the connection between diabetes and dementia. And how you can lower your risks and optimize the health of both your body and your brain. 

What is Type 3 diabetes?

Most people with diabetes don’t have dementia. But they have a higher risk for it over time. People with Type 2 diabetes have a 40% to 60% greater chance of Alzheimer’s dementia. And the risk of vascular dementia is around 90% higher for people with Type 2 diabetes compared to people without diabetes.

In Type 2 diabetes, the cells in the body don’t use insulin effectively. That can lead to organ damage over time. Recent research shows that the same thing can happen to brain cells. Brain cells that have trouble using insulin don’t work as well as they should. That can result in problems with thinking and memory. Type 3 diabetes is a way to talk about the brain changes that happen for some people with Type 2 diabetes. 

Is Type 3 diabetes different from Alzheimer’s disease?

Type 3 diabetes isn’t the same thing as Alzheimer’s disease. But there’s still a connection between the two conditions.

Alzheimer’s disease is the most common cause of dementia. The biggest risk factor for Alzheimer’s is older age. But diabetes is a risk factor as well. 

There’s more to learn about the exact ways that diabetes affects brain cells. But people who have had high blood sugar for longer periods of time have a greater risk of dementia than those whose sugar levels are well managed. People who have more episodes of hypoglycaemia, or low blood sugar, also have a higher risk. 

Most studies on diabetes and dementia have looked at people with Type 2 diabetes, which is much more common than Type 1 diabetes. But one large review found that people with Type 1 diabetes may also have a 50% higher risk of dementia as they age. 

For some people, there may be a shared genetic risk for both Type 2 diabetes and Alzheimer’s disease. One gene, called APOE4, is a known risk factor for Alzheimer’s disease. It turns out that the same gene may affect how the brain uses insulin. 

People with Alzheimer’s disease are also more likely to develop Type 2 diabetes. Interestingly, researchers have also noticed that dementia in some people with diabetes progresses more slowly. And it looks a little different on tests like CT scans and MRIs. This subset of people often have more muscle loss and weakness, and may need to use more insulin. Research in this area may help to develop new kinds of treatment.

How is Type 3 diabetes different from other types of diabetes?

There are four main types of diabetes:

  • Type 1 diabetes happens more often in children and young adults. It’s an autoimmune condition that damages the cells in the pancreas that make insulin. People with Type 1 diabetes have to use insulin every day. 

  • Type 2 diabetes is more common in adults. The body’s cells don’t use insulin well, which leads to higher blood sugar. Type 2 diabetes is treated with diet, exercise, and medications that may or may not include insulin. 

  • Gestational diabetes is high blood sugar that happens during pregnancy. Glucose levels may return to normal after pregnancy. But people with gestational diabetes have a higher risk for Type 2 diabetes later on.

  • Other types that result from taking certain medications or having other medical conditions. These can sometimes lead to high blood sugar. Of note, diabetes that results from a pancreatic disease is sometimes called “Type 3c” diabetes. This isn’t the same as Type 3 diabetes. 

Type 3 isn’t a diagnosed form of diabetes. It’s a way to think about the insulin resistance and damage to brain cells that occur in some people with diabetes.

Can Type 3 diabetes be prevented or treated?

Yes, there are things you can do to prevent Type 3 diabetes. Caring for your diabetes lowers dementia risk.

Things you can do to lower your risks include:

  • Aim for blood sugar levels within the target range: Blood glucose that is too high or too low is risky for brain cells.

  • Manage blood pressure and cholesterol: These conditions often go along with Type 2 diabetes. Managing them also supports brain health.

  • Eat a balanced and nutritious diet: Eating plans like the MIND diet, DASH diet, and Mediterranean diet all have evidence for both diabetes management and brain health.

  • Stay active: Doing regular physical activity helps manage blood glucose and blood pressure. And it improves memory, focus, and mood.

  • Ask about medications: If you have diabetes, talk with a healthcare professional about medications that might also support brain health. Some, including GLP-1s like semaglutide (Ozempic), may help lower the risk of dementia. 

  • Avoid smoking and heavy alcohol use: Smoking and alcohol both increase the risk for Alzheimer’s disease. 

  • Optimize your sleep: Getting at least 7 hours of quality sleep at night helps to protect brain cells.

  • Keep your social connections active: People who have limited contact with others may have an increased risk of dementia.

  • Get support for stress, anxiety, and depression: Treating depression can lower the risk of Alzheimer’s. And it can also improve symptoms for people who have dementia.

The bottom line

Type 3 diabetes isn’t something you’re diagnosed with. It’s a way to recognize and study the links between Type 2 diabetes and dementia. Genetics is a risk factor for both diabetes and dementia. But there are also many risk factors that can be modified. Good nutrition, an active lifestyle, and partnering with healthcare professionals can support the health of your brain and your body.

https://www.goodrx.com/conditions/diabetes/what-is-type-3-diabetes

Tuesday, 2 June 2026

Omega-3 fish oil shows promise against type 2 diabetes

From sciencedaily.com

Fish oil may help switch off inflammation-driven insulin resistance—even in type 2 diabetes patients who aren't overweight

Fish oil may have a surprising role in the fight against insulin resistance, especially in a form of type 2 diabetes that is often overlooked. A Brazilian study published in Nutrients found that omega-3 fatty acids from fish oil reduced glucose intolerance and weakened insulin resistance in rats that were not obese but showed a diabetes-like metabolic condition.

The work was funded by FAPESP and focused on Goto-Kakizaki rats, a well established animal model used to study non-obese type 2 diabetes. Type 2 diabetes is marked by high blood sugar that occurs when insulin, the hormone that helps move glucose from the blood into cells, does not work effectively.


Fish Oil and Insulin Resistance

Omega-3 supplements, including fish oil, are often used by people with cardiovascular disease and type 2 diabetes. However, scientists still know much less about how these fatty acids affect insulin resistance when obesity is not involved.

That question matters because obesity is one of the strongest risk factors for type 2 diabetes, but it is not the whole story. An estimated 10% to 20% of people with type 2 diabetes worldwide are not obese. For these patients, the biological roots of insulin resistance may differ from the better known obesity-linked pathways.

In the study, researchers gave the rats fish oil at a dose of 2 grams per kilogram of body weight (equivalent to 540 mg/g of eicosapentaenoic acid, or EPA, and 100 mg/g of docosahexaenoic acid, or DHA) three times weekly for eight weeks. By the end of the experiment, the treated animals showed lower insulin resistance, better blood sugar control, reduced inflammatory markers, and improvements in several lipid measures, including total cholesterol, LDL ("bad cholesterol") and triglycerides.

The results came from preclinical experiments, so they do not prove that fish oil will have the same effects in people. Still, the findings point to inflammation as a powerful target in non-obese diabetes and suggest that omega-3 fatty acids deserve closer study in this group.

Fish oil rich in omega-3s helped reverse insulin resistance and improve blood sugar control in non-obese diabetic rats. By calming inflammation and reshaping immune cell activity, it revealed a surprising new avenue for tackling type 2 diabetes. Credit: Shutterstock


A Shift in Immune Cells

"Our experiments involved Goto-Kakizaki [GK] rats, an animal model for non-obese type 2 diabetes. We found that insulin resistance can be reduced in these animals by modulating the inflammatory response so as to change the profile of defence cells [lymphocytes] from a pro-inflammatory state to an anti-inflammatory state. This process parallels the response of obese individuals with insulin resistance to omega-3 fatty acid supplementation," said Rui Curi, Director of Butantan Institute's Education Center, Professor of Interdisciplinary Graduate Studies in Health Sciences at Cruzeiro do Sul University (UNICSUL), and coordinator of the study.

Lymphocytes are white blood cells that help direct the adaptive immune response. When their behaviour changes, the effects can spread through the immune system and influence other cells involved in inflammation.

"In previous studies, we observed alterations in both lymphocytes and macrophages [large white blood cells that often reside in adipose tissue and are part of the innate immune system, engulfing and destroying pathogens] in non-obese rats with insulin resistance. In such cases, these cells produce more pro-inflammatory cytokines, as is central in obese people with diabetes," Curi explained.

"The main aim of the study, therefore, was to find out whether supplementation with fish oil [rich in omega-3] could reverse specific alterations in lymphocytes that had been observed in previous research. Our findings increased our knowledge of the link between inflammation and insulin resistance in non-obese animals, confirming that this is a key factor in diabetes even in the absence of obesity," said Renata Gorjão, last author of the article, and Co-Director of UNICSUL's Program of Graduate Studies in Health Sciences.


Inflammation Without Obesity

The Nutrients study, conducted during the PhD candidacy of Tiago Bertola Lobato, was part of a broader FAPESP-supported project exploring how insulin resistance develops in non-obese animals.

Curi noted that obesity is a major diabetes risk factor, but not the only one. In people who develop diabetes without obesity, one leading hypothesis is that genetic factors may play an important role. In another study published in Cells, Curi, Gorjão, and colleagues investigated whether delayed intestinal transit might also contribute to insulin resistance in non-obese individuals.

"Most obese people have chronic low-level inflammation, which is known to affect the insulin signalling pathways. Adipose tissue, which is augmented in obesity, releases pro-inflammatory cytokines that affect the insulin signalling pathways, promoting insulin resistance. In the non-obese model, this impactful characteristic of adipose tissue is absent, but systemic inflammation is present," Curi said.

The group had previously shown systemic inflammation in non-obese GK rats with insulin resistance in a study published in the International Journal of Molecular Sciences.

Another paper from the same project reported that anti-inflammatory defences appear to break down early in non-obese GK rats with insulin resistance. Lymph nodes (part of the immune system) from newly weaned 21-day-old GK pups already showed reduced markers of regulatory T-cells (Tregs, cells with anti-inflammatory characteristics). The researchers also detected other early inflammatory changes. That work was published in FEBS Letters, a journal of the Federation of European Biochemical Societies.


How Omega-3s May Help

The Nutrients study suggests that fish oil may work by moving immune activity away from a damaging inflammatory pattern and toward a more protective one.

"Fish oil supplementation reversed this pro-inflammatory profile, displaying a significant anti-inflammatory effect and reducing polarization of Th1 and Th17 cells [lymphocyte subtypes that perform crucial functions in inflammation], followed by a rise in the percentage of Tregs, which can inhibit the activation of pro-inflammatory lymphocytes. Thus the action of omega-3 fatty acids on lymphocytes, modulating them from a pro-inflammatory state to an anti-inflammatory state, may have triggered the reduction in insulin resistance in these animals," Lobato said.

That immune shift is important because insulin resistance is not only a problem of sugar metabolism. It is also deeply connected to inflammation. When inflammatory signals remain elevated, they can interfere with insulin signalling and make it harder for cells to respond to the hormone.

The study adds to a growing view of type 2 diabetes as a disease shaped by both metabolism and the immune system. In this case, fish oil appeared to improve blood sugar regulation not simply by changing fat levels, but by changing the inflammatory environment that helps drive insulin resistance.


What Later Studies Add

Since the Nutrients paper was published, related human research has continued to examine how omega-3 fatty acids may influence early diabetes risk and metabolic health.

A 2025 double blind randomized controlled trial in Food and Function tested fish oil supplementation in healthy middle aged and older adults. Over 12 weeks, the fish oil groups had dose related increases in serum EPA and DHA. The researchers also reported decreases in fasting insulin and the HOMA-IR index, a common marker of insulin resistance. Fasting blood glucose trended downward across groups, and several lipid related measures also improved.

Another 2024 analysis in Nutrition and Diabetes used modelling data from 161 patients with type 2 diabetes to explore the relationship between omega-3 levels and HbA1c, a longer term marker of blood sugar control. The authors reported a dose related association and proposed that omega-3 intake could be studied in a more individualized way, while also noting that the role of omega-3s in type 2 diabetes remains debated.

Together, these studies do not settle the question of whether fish oil should be used to manage diabetes. Human evidence remains mixed, and the Brazilian study was conducted in animals, not people. However, the newer findings are consistent with the idea that omega-3 fatty acids may affect insulin resistance and inflammation in ways worth testing more carefully.


More Research Still Needed

Despite the promising findings, the researchers stressed that the results should be interpreted cautiously. Animal studies are useful for uncovering biological mechanisms, but clinical trials are needed before scientists can know whether the same strategy works in people with non-obese type 2 diabetes.

"These studies involved well-established experimental models that mimic insulin resistance in non-obese individuals. Trials in humans are needed to estimate the ideal dose and the most indicated type of omega-3 fatty acid," Curi said.

For now, the study offers a compelling clue: in diabetes, body weight may not be the only driver of insulin resistance. Inflammation can play a central role even without obesity, and fish oil may help reveal how that hidden process can be changed.

https://www.sciencedaily.com/releases/2026/05/260530004626.htm